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Are the lasting effects of acne more than just skin

Are the lasting effects of acne more than just skin deep?

The teen years are a trying time for any person. Although the worries and cares of adulthood are still far into the future, the teen is not exactly a happy and relaxed person. The carelessness and ignorance of childhood makes room for rebellion and questions about ones values and place in the world. Looking for their own road through life teens feel insecure and, often enough, misunderstood. Unfortunately, if the search for a place under the sun is not enough, teenagers also have to deal with other problems, such as acne. This unsightly condition is the bane of many teenagers because of the effects it has at psychological level.

It doesnt take a lot to foster a negative self image in a teenager. Most of them are at least somewhat insecure about their outward appearance because of various real or imaginary blemishes. But a real condition that causes true discomfort and mars a young face is something else altogether. The fragile self image and self respect of teenagers are hard pressed to cope with the pressure exerted by the opinions of others and the comparisons with various role-models that teenagers find for themselves. In extreme cases, the negative self image turns into self hatred and the disfiguring condition is seen as a cruel instrument of self punishment for failing to rise up to some standard or other.

Teenagers live in small worlds. Parents, friends and the amorphous group of half-familiar school mates are the limits of their social lives. Naturally, teenagers tend to attach more importance to the praise or criticism coming from their friends or school mates, on grounds that parents love you anyway and are fatally biased. But, since teens can be very cruel to each other, the criticism of school mates is frequently unkind and meant to hurt. This serves to increase the feelings of anxiety, insecurity and self hatred, resulting in withdrawal from the social environment into a private world of pain and shame.

Teenagers are terribly earnest about outward appearances and criticism. They are still away from the age when human beings come to terms with themselves and are no longer much interested in what others say or think. Hiding ones body or face and feeling ashamed just because somebody else said that this is the thing to do is a mistake and teenagers should be helped to see this. Acne can be defeated with persistence and by using the right products. However, the psychological effects of acne must also be fought, or else they may never go away. The teenager who hated himself for having pimples on his face will turn into the adult who hates himself for being a little overweight or not making as much money as some co-worker.

The feeling of discomfort and shame with oneself does not always go away in time. Sometimes it just finds a new problem to act as its power source. This is the lasting effect of acne: a poor self image, a lack of confidence and a feeling of being at a disadvantage when comparing oneself to other people. These moods and mindsets can ruin anybodys social life and often they also get in the way of professional development. Unfortunately, acne is not just skin deep.

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The Men On Steroids May Lose Libido & Grow Impotent

The Men On Steroids May Lose Libido & Grow Impotent

The men on steroids may experience temporary sterility, testicular atrophy, and the development of female sex characteristics. Testosterone, the male sex hormone, is responsible for the growth of sexual organs. Testosterone is secreted in male testes.

The amount of testosterone in the male bloodstream is controlled by the pituitary gland in the brain. If testosterone level is low, the pituitary gland secretes the hormones FSH (“Follicle Stimulating Hormone”) and LH (“Luteinizing Hormone”) that make testes to secrete more testosterone, but if testosterone level high, the pituitary gland drops FSH and LH, and testosterone level drops consequently. This way body regulates the testosterone production.

In the men on steroids, finding high quantity of anabolic steroids that are chemically similar to testosterone, the pituitary gland cuts down the testosterone production in the testes. Hence the men on steroid make their testes remain inactive or not producing testosterone as long as they go on taking steroids.

This results in the lowering down the production of sperm cells, which grow to abnormal shapes in the men on steroids. If the men on steroids continue abusing anabolic steroids for long, they may become infertile, but if they stop taking steroids, their testicular function will probably return to normal. The long term men on steroids have lasting sterility and loss of libido with impotence due to a permanent effect on the pituitary.

The levels of testosterone is 30 % more in men, that is why men normally have erections or feel like having sex in morning. The balance of testosterone gets disturbed in the men on steroids, who may lose their libido or grow impotent.

The men on steroids get lots of anabolic steroids accumulated in their bodies. The large amounts of anabolic steroids get metabolized to form conjugated estradiol and estrone, female hormones. This results in the breast growth in the men on steroids. The condition is medically termed as gynecomastia. This is an irreversible side effect of anabolic steroid abuse and may accumulate from one cycle of use to the next. The body changes a portion of the anabolics to conjugated estradiol and estrone, female sex hormones, by a chemical process called aromatisation.

Cycle Of Hair Growth And Information about Follicles.

At any Stage of our life, only 10% of hair is in resting phase. They Fall off in 2-3 months and the new hair grows in a total time of 2-6 years. About 90% of the hair grows on our scalp at a time and They grow at a rate of 1 cm. per month. … Continue reading Cycle Of Hair Growth And Information about Follicles.

At any Stage of our life, only 10% of hair is in resting phase. They Fall off in 2-3 months and the new hair grows in a total time of 2-6 years. About 90% of the hair grows on our scalp at a time and They grow at a rate of 1 cm. per month.
Normally hair lasts for two to four years in men and four to six years in Women.

Hair Internal Structure : Deep Inside The hair follicle, the hair forms inside a hair bulb and protrube and grow outside.
Any Method of hair improvements like shampooing, conditioning, cutting, sun exposure does not effect the rate of grwth of the hair.

There Are 3 phases of hair growth –
1. Anagen which takes a time of about 1000 days or 3 years.
2. Catagen lasting for 10 days.
3. Tologen for nearly 3 months.

Anagen includes the start of growth of hair and Tologen is the end i.e. trhe shedding phase of hair. The Hair bulb goes on coming outside from the start to the shedding phase. Hair groth is effected by the seasonal changes, i.e. hair grows more quicker in winter than in summer as a result of the seasonal change. In the Catagen phase, Hair growth is stopped for a small time and no pigment is produced at this time.

The time of Anagen Phase is usually fixed and and is determined genetically and is also responsible for the length of the hair. The no. of Hair Follicles in a human head counts approximately 100,000. Each Follicle produces hair for about 20 times in the lifetime. In a new born baby, the hair follicles grow hair in a Unison, i.e. all at a same time but as time passes, the follicles produce hairs at different times.

If a hair is Plucked from the head, the follicle is not ruptured but it starts to produce a new hair. As age increases, the shedding of follicles is evidently seen in most of the people in the top of the head and also in the forehead. The Hair does not grow in a definite straight manner but makes the follicle to stand in some constant angle. Depending on this angle, the Hair are always set to lie. The Stream is usually in a twisted manner but it is then influenced by the way of combing of hair by people.

Finally, Hair grows at a slow rate, so utmost care is needed to prevent them from shedding.

Intracerebral Hemorrhage: Bleeding Inside the Brain

All strokes damage the brain by disrupting circulation, but strokes come in multiple varieties. Because different parts of the brain are specialized to perform specific functions, symptoms produced by strokes vary according to what part of the brain was injured. In one patient the symptom might be weakness on one side of the body. In another it might be a partial loss of vision. In still another, a loss of speech. And symptoms can vary in intensity from mild to severe according to how large the area of damage is and whether it occurred in a pivotal location.

Strokes can also vary according to another fundamental difference — whether they involve a blocked blood vessel or a hemorrhage. Most strokes are due to the former in which brain-tissue damaged by lack of circulation is called an infarction. But 10-15% of strokes involve bleeding from ruptured blood vessels within the brain tissue, and while it’s bad enough to have an infarction, hemorrhagic strokes (intracerebral hemorrhages) can be even more devastating.

One prominent figure with spontaneous intracerebral hemorrhage is Ariel Sharon, whose hemorrhagic stroke occurred while he was still prime minister of Israel. Although some patients with intracerebral hemorrhage recover to a point of being able to enjoy other people and regain some independence in functioning, Sharon’s poor clinical outcome is all too common in patients with this disease.

The additional problem with hemorrhagic strokes is that the new deposit of blood occupies space — sometimes a lot of it — and there is only so much space within the skull (braincase) to go around. The fresh hemorrhage crowds and distorts the brain-tissue next to it, and additionally subjects the rest of the brain to increased pressure that can itself be damaging. Because of these distortions and pressure-changes, a patient with intracerebral hemorrhage often shows a decreased level of consciousness or even coma.

Another kind of spontaneous bleed within the braincase is subarachnoid hemorrhage, often caused by ruptured aneurysms outside the brain but inside the braincase. While this, too, is a very serious condition, it is not the focus of this particular essay, and spontaneous intracerebral hemorrhages are not caused by aneurysms of this kind. Yet another kind of bleed that can be confused with (primary) intracerebral hemorrhage is secondary hemorrhage. This occurs in some patients who started out with infarctions of the brain but who had subsequent bleeding from fragile blood-vessels around the infarction’s edges. This kind of bleed is not quite as serious as that which occurs when the bleed is primary (the initial event).

How are intracerebral hemorrhages diagnosed? Since the 1970s when computed tomographic (CT) scans were introduced, this imaging technique has been the most effective and sensitive tool. A fresh hemorrhage within brain tissue is dramatically evident on CT scans. And unlike infarctions that can take a day or two to show up on CT scans, hemorrhages are already visible at the earliest moment a scan can be made.

Although surgical removal of blood-clots from the surface of the brain — called subdural and epidural hematomas — can be life-saving and function-sparing, surgery for a bleed (hematoma or blood-clot) within the brain tissue itself is another story. Some studies comparing outcome between operated and unoperated patients with intracerebral hemorrhage showed improved outcome, on average, for operated patients, while still others showed worsened outcome. Operated or unoperated, patients had high rates of death and disability.

Because of the limited prospects for meaningful improvement, surgery for intracerebral hemorrhage is often an act of desperation. One crusty old clinician was blunt about the direness of the situation, saying, “Show me a patient with intracerebral hemorrhage whose life was saved by surgery, and I’ll show you a patient you wish you hadn’t operated on.” His point was that survivors of this operation usually show severe impairments.

However, one form of hemorrhage within brain tissue is probably a special case, and that is hemorrhage within the cerebellum, located within the bony braincase just above the nape of the neck. Surgical extraction of blood clots occurring within the cerebellum prevents excessive pressure on the nearby brainstem that handles a lot of basic and necessary functions, like breathing.

Administration of cortisol-type steroids is a nonsurgical treatment that has been studied in a scientific way, comparing treated patients to untreated patients with the same condition. The steroids didn’t help. Decreasing the patients’ blood pressures by administering medication has likewise been studied, but with the same outcome — no benefit. However, in a preliminary study one nonsurgical treatment showed promise. Intravenous administration of activated factor VII (a natural component of the blood-clotting system) reduced expansion of the intracerebral blood-clot, death and disability when given within four hours of the initial hemorrhage. A larger study is underway to see if this benefit holds up under further analysis.

Otherwise, what can be done acutely for this condition? Individualizing treatment seems rational, even if unproved. For example, if the patient had a bleed while taking a blood-thinner (as was the case with Ariel Sharon) then it makes sense to stop the blood-thinner or reverse its effects. Supportive management, like administering intravenous fluids to prevent dehydration, monitoring for irregular heartbeats and protecting the patient’s airway also make sense. If the patient can’t consume food in the usual way, feeding through tubes or intravenous lines can be considered, though this decision can be postponed until the patient’s prospects are more apparent.

Who is at risk for intracerebral hemorrhage? Neurologists at Malmo University Hospital in Malmo, Sweden, compared 147 patients with intracerebral hemorrhage with 1029 similar but stroke-free patients in order to determine risk factors. They found that hypertension (high blood pressure), diabetes, elevated triglyceride levels in the bloodstream, history of psychiatric problems, smoking and (surprisingly) short stature were more frequent in patients with intracerebral hemorrhage.

However, when it comes to modifiable risk-factors (those that one can do something about) a variety of studies indicate that hypertension is the single most important factor. Thus, treatment of hypertension, when present, is probably the single most effective thing that one can do in order to prevent this disease.

(C) 2006 by Gary Cordingley